Mechanism of tubulointerstitial damage by proteinuria and hypertension

Kidney cartoon


The results of these pathological processes (proteinuria and hypertension), which generate tubular interstitial damage through apoptosis induced by proteinuria and glomerular sclerosis with tubular interstitial fibrosis due to barotrauma, endothelial dysfunction, vasoconstriction and chronic hypoxia secondary to high blood pressure.

In recent years the lines of research generated around chronic kidney disease has emphasized molecular, genetic and ultra-structure aspects, looking for new biomarkers of damage and progression of the disease, equally, computerized systems have been developed which combine multiple clinical variables, trying to identify those patients with kidney disease who remain stable or progress to more advanced stages.


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